Hong Kong Med J 2014;20:52–8 | Number 1, February 2014 | Epub 9 Dec 2013
DOI: 10.12809/hkmj134146
© Hong Kong Academy of Medicine. CC BY-NC-ND 4.0
REVIEW ARTICLE
Current management of acute scaphoid fractures: a review
Jason PY Cheung, MB, BS, MMedSc;
Chris YK Tang, MB, BS;
Boris KK Fung, FRCS, FCRCE (Ortho)
Department of Orthopaedics and Traumatology, University of Hong Kong
Medical Centre, Queen Mary Hospital, 102 Pokfulam Road, Hong Kong
Corresponding author: Dr JPY Cheung (jcheung98@hotmail.com)
Abstract
The aim of this review was to present currently available evidence on the management of
acute scaphoid fractures. Acute scaphoid fractures are usually diagnosed by a combination of
history, physical examination, and radiography. However, in many patients scaphoid fractures
are still missed. Thus, the general trend is to over-treat patients with a suspicion of scaphoid
fracture. Many aspects of scaphoid fracture management are still controversial and different
institutions vary in their approach.
Introduction
Scaphoid fractures have been extensively investigated in the past. They are the most
common type of carpal fractures and are usually found in young men,1 accounting for
2 to 7% of all fractures, and 70 to 80% of carpal fractures.2 Scaphoid fractures affecting
the waist (70%) are the commonest type in adults, followed by distal pole fractures (10-20%), proximal pole fractures (5-10%), and tubercle fractures (5%).2 Conversely, 52% of
all scaphoid fractures in children involve the tubercle, 33% affect the distal third, and
15% affect the waist,2 though this discrepancy may be partially accounted by the fact that
children’s scaphoids are not fully ossified in their proximal pole, making a waist fracture
look like a proximal pole fracture. Scaphoid fractures are commonly prone to complications
due to delayed treatment or misdiagnosis. Avascular necrosis is particularly common, with
estimated rates of 13 to 50%.3 Other complications such as nonunion, malunion, carpal
instability, and radiocarpal arthrosis are also frequently seen. Thus, early diagnosis and
treatment are critical for a better prognosis.
Acute scaphoid fractures can be difficult to diagnose. According to some studies,
the prevalence of true fractures among patients with suspected scaphoid fractures
may only be 5 to 10%.1 Multiple radiographs may not be able to pick up all scaphoid
fractures and consequently, clinicians attempt to avoid undertreatment by liberal use
of cast immobilisation. However, overtreating patients results in a loss of work days and
productivity and increased health care costs,1 and it is conjectured that 76 to 100% of such
cases undergo inappropriate initial immobilisation.4
Undisplaced scaphoid fractures are not benign injuries and warrant prolonged
plaster cast immobilisation or early osteosynthesis. Clinicians should have a high index
of suspicion and must be meticulous in studying the clinical examination findings and
radiographs.
Anatomy
The scaphoid forms the bridge to the distal carpal row (trapezium, trapezoid, and capitates)
and the proximal row (proximal pole to the lunate) by a ligamentous network including
the scapholunate interosseous ligament and extrinsic palmar ligaments. The scaphoid can
be simply divided anatomically into the proximal pole, the waist, and the distal pole. Its
surface is mostly (80%) covered by articular cartilage.5 The articular surfaces include the
proximal pole articulation with the radius and lunate and the distal pole articulation with
the capitate, trapezium, and trapezoid.
Being predominantly articular, the blood supply to the scaphoid has limited access.
Branches from the radial artery (dorsoradial arteries) form arches at the dorsal wrist capsule
and enter the scaphoid at its dorsal ridge. This provides 70 to 80% of the blood supply to
the scaphoid.3 5 The other 20 to 30% are supplied by the superficial palmar arch or branches
of the radial artery that reach the distal palmar area of the scaphoid.3 5 Thus, the proximal
pole depends solely on intraosseous blood flow and fractures here have a high risk of
osteonecrosis associated with a prolonged period of healing. An average of 3 to 6 months are required for healing in these fracture types and
nonunion is quoted to ensue in 5 to 10%.3 Additional
nutrient arteries supply the distal pole via the area of
the scaphotrapezium ligamentous attachment.
Biomechanics
The usual mechanism of injury is a forced
dorsiflexion wrist injury such as a fall on outstretched
hand. Cadaveric studies have shown that fractures
occur when the wrist is kept in 95 to 100 degrees of
extension and a dorsiflexion load is applied to the
radial half of the wrist with the radioscaphocapitate
ligament kept as the fulcrum.5 Failure in compression
occurs on the dorsal side of the bone and failure in
tension on the palmar side. Dorsal angulation of the
fracture is caused by opposing rotational moments
on the proximal and distal poles of the scaphoid.
Furthermore, dorsal intercalated segmental
instability (DISI) ensues if the proximal carpal row
is in extension. Bending forces to wrist fractures are
resisted by intact scaphoid-carpal ligaments.6 Distal
pole and tubercle fractures are due to direct impact
and forced ulnar deviation causes avulsion fractures
at radial collateral ligament attachments.
Assessment
Classical examination findings of tenderness at the
anatomic snuffbox and the volar aspect of the distal
tuberosity and positive scaphoid compression test
(pain on axial compression of the thumb metacarpal)
raise suspicions that warrant further investigation.
A study by Unay et al7 suggested that pain during
thumb-index pinching (sensitivity 73%, specificity
75%, positive predictive value 96%, and negative
predictive value 23%) and pain during forearm
pronation (sensitivity 79%, specificity 58%, positive
predictive value 82%, and negative predictive value
54%) aid the diagnosis of scaphoid fractures, but are
absent in 27% of cases. Overall, the specificity for
clinical examination shown in the literature was only
74 to 80%3 and the mean positive predictive value
was quoted to be only 21%.8 Other physical findings
that may help to diagnose scaphoid fractures include
limitation in end arc of motion with flexion and radial
deviation, and reduced grip strength.
Nonunion can be found in up to 12% in cases
of missed scaphoid fractures.3 Therefore, scaphoid
fractures must be identified early and immobilised
appropriately. Imaging techniques can aid in the
diagnosis of occult fractures. Plain film radiography
can detect a fracture in 70 to 90% of cases.8 Four views
are necessary: posterior-anterior (PA) wrist, lateral
wrist with extended fingers, anterior-posterior (AP)
wrist with flexed fingers (scaphoid lies parallel to
the film with flexed fingers), and the wrist in 25 to
45 degrees supination with flexed fingers.9 Neutral or
ulnar-deviated PA films do not show waist fractures
well, because the axis of the scaphoid is flexed towards
the beam and the tubercle overhangs the body.
Fractures of the dorsal sulcus are best demonstrated
on a 45-degree PA oblique view, and proximal pole
fractures on a 45-degree AP oblique view.2 Scaphoid waist fractures are best seen on an ulnar-deviated
PA view with 20 degrees of elbow flexion.2 The
semipronated oblique view visualises the waist of the
scaphoid best, but multiple views such as PA, lateral
and ulnar-deviated and clenched-fist views may be
required to make a correct diagnosis.9 Lateral X-rays
may only detect tuberosity and distal third fractures,10
but are also essential to show the carpal alignment
and distal radioulnar joint alignment. A proper view
should show a co-linear capitate and radius, with
the pisiform located between the distal pole of the
scaphoid and the body of the capitate.3
Other adjuvant imaging techniques may be
required to diagnose scaphoid fractures. Computed
tomography (CT) is usually used to identify fractures
and nonunions and for preoperative planning, and it
is better for detecting occult fractures of the cortex
with a mean sensitivity of 94% and specificity of 96%.1
It was found to have a mean negative predictive value
of 99% in a study by Ty et al,11 which means it is very
unlikely to miss a scaphoid fracture. Furthermore, CT
is readily available in urgent care settings and is more
cost-effective than magnetic resonance imaging
(MRI).
Magnetic resonance imaging has a mean
sensitivity of 98% and specificity of 99%.1 It can locate
trabecular fractures and help identify other causes of
wrist pain if a fracture is not found, besides helping
to determine the vascularity of the proximal pole
preoperatively. It is especially useful in diagnosing
proximal pole fractures, which may develop
avascular necrosis. Acute fractures show normal or
decreased T1 and increased T2 intensity.3 Nonunion
and impaired vascularity are often seen with low
T1 and T2 marrow signal intensity which correlates
with poor healing.3 Notably, MRI is more sensitive in
detecting occult scaphoid fractures, with fewer false-positives
than bone scans.3 Thus, it can accurately
exclude patients without scaphoid fractures and
facilitate discontinuing immobilisation. For planning
the management of cases of scaphoid nonunion, MRI can be used following internal fixation, as the bone
marrow signal can be assessed even in the presence
of a titanium alloy screw.12
Scaphoid fractures are commonly associated
with injuries to the carpal ligament or triangular
fibrocartilage complex, and reported in 35% of
affected patients and intercarpal soft tissue injury may
ensue in 86% of instances.13 Usually these conditions
can be treated conservatively as mild ligament tears
heal without long-term complications. However,
carpal ligament injuries may lead to symptomatic
chronic carpal instability. Thus, surgical fixation and
early mobilisation may be indicated in more severe
cases. In the most serious cases, scaphoid fractures
can constitute part of the abnormality in perilunate
or lunate dislocations of the wrist. Identifying and
assessment of these injuries can be performed with
a wrist arthrogram or arthroscopy. Besides soft tissue
injuries, distal radial fractures are also found quite
commonly due to shared mechanisms of injury.
Classification
Herbert’s classification system is the most well-known
and commonly used, as it defines stable and unstable
fractures. Type A fractures are stable acute fractures;
type B are unstable acute fractures; type C are delayed
unions (>6 weeks of plaster immobilisation), and type
D are established nonunions (fibrous or sclerotic).
Stable fractures include fractures of the tubercle
(A1) and incomplete fracture of the waist (A2). Type B
fractures are acute unstable fractures. These include
subtypes B1 (oblique fractures of the distal third of
the scaphoid), B2 (displaced or mobile complete
fractures of the waist), B3 (proximal pole fractures),
B4 (fracture dislocations), and B5 (comminuted
fractures).14
Treatment
Treatment of acute scaphoid fractures is controversial
and each centre has different criteria for conservative
versus operative treatment. In patients with a
suspected fracture but no obvious findings on
X-rays, most centres advocate joint immobilisation
before repeat imaging for reassessment at a later
time. However, casting makes exclusion of fracture
and determination of fracture union by follow-up
X-rays more difficult, for which reason follow-up
CT may be warranted. Fixation is suggested if the
MRI shows a proximal pole fracture.3 Since cartilage
covers 80% of the scaphoid, no fracture callus can
be made to stabilise the fracture site during healing6
and thus rigid fixation is mandatory. Healing on the
X-ray is inferred from disappearance of the fracture
line, spot welding between fracture fragments, or
callus formation. With these findings, immobilisation
can be discontinued and patients can be allowed a gradual return to activities.
Treatment depends primarily on the location
and degree of displacement. Distal pole fractures are
usually due to avulsion of the scaphoid tuberosity
or impaction of the distal articular surface. These
fractures have a good vascular supply that enables
rapid healing in 4 to 6 weeks with a short arm thumb
spica cast.5 Traditionally, undisplaced, stable waist
fractures are treated in short-or long-arm casts.
These often involve prolonged immobilisation of
up to 12 weeks.3 Union can be achieved in greater
than 90% of affected individuals.5 However, recent
evidence suggests improved results with operative
fixation. Prolonged immobilisation disrupts collagen
homeostasis resulting in loss of normal connective
tissue characteristics, allowing tendons to glide and
the joint capsule to stretch.
Non-operative
Some studies suggest a further 6 weeks of
immobilisation before offering operative fixation if at
6 weeks the CT shows an unhealed fracture.3 Others
suggest that most fractures of the scaphoid waist
unite after 8 weeks of immobilisation but may require
as long as 12 weeks.15 Overall scaphoid waist fractures
can unite satisfactorily in 85 to 95% of patients.16
Scaphoid fractures are generally immobilised in a
scaphoid cast (proximal phalanx of thumb in palmar
abduction leaving the interphalangeal joint free)
or a Colles’ cast (exposing the thenar eminence
and leaving the metacarpophalangeal joint free).
Pinch grip function is impaired in scaphoid casts;
the Colles’ cast allows for greater overall range of
thumb movement and improved function.17 The
fine pinch grip of the thumb should be preserved
as much as possible, for which Colles’ casting rather
than scaphoid casting offers better preservation of
function.17 However, there are no data on the longterm
results of fracture healing following these two
types of casting.
The wrist position during immobilisation has
also been investigated. Hambidge et al18 showed
that the frequency of nonunion was not influenced
by the position of immobilisation (P=0.46) and 108 of
121 fractures united after 12 weeks of immobilisation.
However, wrists immobilised in 20-degree flexion
results in less extension at the 6-month follow-up.18
A possible explanation was that immobilisation in
flexion may have produced increased flexion stress
on the fracture, causing a humpback deformity, which
restricts wrist extension and causes persistent pain.18
An alternative explanation for restricted extension
could be related to soft-tissue injury, joint adhesions,
or contracture of the palmar capsule at the wrist.18
Immobilisation in a Colles’ cast with the wrist in 20-degree extension is therefore recommended.18
Long-arm versus short-arm casting is
controversial. Biomechanical studies in cadavers
show fracture site motion during forearm rotation.5
Forearm rotation leads to excessive scaphoid fracture
motion, which may impair bone healing. Excessive
motion is an indication for long-arm casting to
restrict forearm rotation and to reduce the associated
displacement of bone fragments. Displacement of
more than 1 mm is associated with instability and is an
indication for open reduction and internal fixation.19
A cadaveric study on scaphoid waist fractures by
Kaneshiro et al20 showed that significant fracture
site motion could occur with forearm rotation in a
short-arm thumb spica cast. Some forearm rotation
may even occur when long-arm casting is used, but
the displacement should be less than 0.5 mm.20 Thus,
long-arm casting is recommended.
As mentioned above, most studies suggest
immobilisation for 8 to 12 weeks for scaphoid waist
fractures.15 16 17 18 21 Geoghegan et al15 showed that 89% of
undisplaced scaphoid fractures achieved union by
4 weeks and mobilisation could begin at that time.
Allowing mobilisation with a wrist splint at week 4
can reduce the period of disability associated with
nonoperative treatment.15 Böhler et al9 showed a
96% healing rate in 580 undisplaced scaphoid waist
fractures with 6 weeks of immobilisation with a simple
unpadded dorsal fist plaster splint that includes the
thumb. The hand needs to be kept in neutral position
when using the dorsal plaster splint.9
Nonoperative treatment is successful in
achieving union but there are disadvantages of
immobilisation, namely stiffness, diminished grip
strength, and delayed return to work. Pseudoarthrosis
ensues in approximately 4% of patients who only
have casting, and is usually associated with vertical
oblique fracture patterns (due to tilting and shearing
forces) and diastasis between bone fragments.9 Young
and active patients are unlikely to tolerate several
months of immobilisation due to the pressures of
work or athletics. Currently, therefore, there is a
trend towards operative management to reduce the
number of days of inactivity.
Operative
In theory, early internal fixation has the benefits
of early return of wrist movement, a higher rate
of union, an early return to work and sport, and
avoiding the need for a plaster cast. Reduction of
the fracture in anatomical alignment is vital for
good results. If reduction cannot be achieved by
closed means, open reduction is necessary. Usually
simple hyperextension of the wrist can achieve good
reduction. In addition, Moser et al22 suggested having
the arm in extension during surgery to maintain the
reduction. Percutaneous fixation of the fracture limits
the risk of devascularising fracture fragments and protects the ligaments and volar capsule. However,
for percutaneous fixation to be feasible, the fracture
must not be displaced or reducible by closed
means. On the contrary, there is no controversy
about treating displaced scaphoid fractures by open
reduction and internal fixation.5 Surgical stabilisation
allows the patient to perform early range of motion
exercises and avoids prolonged immobilisation.
In the literature, evidence in favour of surgery
is not overwhelming. A meta-analysis by Bhandari
and Hanson23 showed that internal fixation resulted
in a significantly earlier return to work (by 8 weeks) as
compared with casting. However, both methods did
not differ in terms of outcomes such as grip strength
(P=0.24) and range of motion (P=0.67).23 Furthermore,
the risk of nonunion was also found to be similar
(P=0.28).23 Similarly, Saedén et al21 showed that a
follow-up period of up to 12 years after the fracture
revealed no difference in pain or discomfort between
the operative and conservative treatments. Dias et
al24 had similar results in terms of grip strength and
range of movement after follow-up for 93 months.
However, McQueen et al25 showed that percutaneous
screw fixation attained quicker union (9 vs 14 weeks,
P<0.001) for treatment of Herbert types B1 and B2
fractures of the scaphoid waist.
The two usual approaches for percutaneous
fixation of scaphoid fractures involve volar traction
assistance and the dorsal minimal incision (manual
reduction with a guidewire as a joystick technique
or arthroscopy-assisted reduction). In the volar
technique, the wrist is extended over a towel roll
to allow proper insertion of the guidewire. Yip et
al26 suggested the 45-degree supination oblique
view when determining the length of the screw and
avoiding over-penetration into the radioscaphoid
joint space. The headless screw must be fully buried
beneath the articular cartilage of the proximal
scaphoid, so as to avoid radioscaphoid impingement.
Scaphoid fixation is best accomplished with the
longest screw placed in the distal scaphoid poles.6
Bone density is greatest in the scaphoid poles, where
it provides the best fixation.6 Fractures of the distal
two thirds can also be approached volarly, as this
approach avoids injury to the dorsal blood supply.
The volar technique is contra-indicated in proximal
pole and oblique fractures, as the screw cannot
cross the fracture line perpendicularly to obtain
adequate compression and purchase.27 This leads
to displacement of the fracture. During surgery,
the scaphoid is in a flexed posture relative to the
longitudinal alignment of the distal radius. From
the volar approach, the proximal point to aim at
is the proximal ulnar corner of the scaphoid at the
insertion of the scapholunate ligament.28 The starting
point of the surgery is at the scaphotrapezial joint
through the proximal thenar muscles. Besides a small
terminal branch of the radial nerve, the operation is at a safe distance from the median nerve motor
branch and from the radial artery. The drawback of
volar surgical approaches is the difficulty in obtaining
fracture reduction, which may therefore result in
nonunion of proximal scaphoid pole fractures.6 The
trapezium is in a position that blocks wire placement
volarly, and therefore placing a guidewire along the
central scaphoid axis is difficult such that the screw
can also penetrate the joint.6
For the dorsal approach, the distal point aimed
at is the centre of the scaphotrapezial joint or the
base of the thumb. Thus, this allows for a more
central placement in the distal pole.28 The dorsal
approach provides direct unobstructed access to the
proximal pole permitting the placement of a central
axis guidewire for screw implantation. There is better
fracture fixation as the purchase of the screw threads
in the proximal fragment tends to be greater.29
However, the disadvantages of this technique include
poor exposure to the distal third of the scaphoid,
damage to the articular cartilage of the proximal pole
of scaphoid, potential entrapment of the extensor
tendons, damage to the dorsal ligaments, and risk
of vascular injury.29 30 Moreover, to insert the screw
through the most proximal part, the wrist has to
be fully flexed during the procedure.29 Flexing the
wrist may cause the distal fragment to adopt a flexed
posture and cause the proximal fragment to follow
the lunate into an extended posture, producing the
hump-back deformity.29 Distal pole fractures can
present technical difficulties for insertion of a volar
screw perpendicular to the fracture line and are
therefore best suited for fixation using the dorsal
fixation technique.27
Arthroscopy can assist fracture reduction, and
real-time mini-fluoroscopy can guide the dorsal
percutaneous insertion of a headless compression
screw.6 Arthroscopic examination can also permit
assessment of concurrent ligamentous injuries
and demonstrate osteochondral fragments at the
midcarpal row.6 Thus, arthroscopic-assisted surgery
can treat both scaphoid fractures and carpal ligament
injuries.31 Arthroscopy also preserves the key
ligaments and blood supply, allowing for immediate
hand rehabilitation.32
Complications
Up to 20% of patients may endure residual pain
despite a normal grip strength and wrist movement
after surgery.33 These persistent symptoms can be
due to intra-operative articular cartilage damage.
Damage affects the distal scaphoid-radial styloid
joint first, and later progresses to the scaphocapitate
and capitolunate joints. Osteoarthritis occurred in
5% of patients with a history of a scaphoid fracture
despite normal healing,34 but usually did not present
until several decades after injury. The opening of the scaphotrapezial joint for screw insertion
during surgery may result in the development of
osteoarthritis.21
Malunion of the scaphoid may produce a flexion
(humpback) deformity with ulnar deviation and
pronation of the distal fragment. Flexion deformity
within the scaphoid causes loss of extension at the
radiocarpal and midcarpal joints. Amadio et al35
showed that only 27% of patients with interscaphoid
angles of >35 degrees had satisfactory results in
terms of pain, function, movement, and strength. This
contrasts with 83% of the patients having satisfactory
results with interscaphoid angles of <35 degrees.35
It is accepted that for a scaphoid fracture, 6
months must elapse before a diagnosis of nonunion
can be made. Such patients endure pain and
poor function; in 35% lateral wrist X-rays yielded a
humpback deformity (due to flexion angulation
between the proximal and distal scaphoid poles)
of patients,4 and 42% showed DISI.4 In a review of
104 patients with symptomatic nonunion, all of
them developed osteoarthritis.36 Fibrous union is
visualised as irregularity at the fracture line, while
in pseudoarthrosis the two bone halves move
independently causing articular damage to the
radial facet.33 Displacement of the nonunion with
the incongruent cartilaginous surfaces and carpal
instability also contributes to the development of
osteoarthritis.4 Many now advocate internal fixation
and the use of bone grafting for the treatment
of established nonunion. Bone healing usually
occurs (in about 75% of cases) but there may be
persistent humpback deformity (16%), associated
DISI deformity (12%), and osteonecrosis (4%).4
Nevertheless, late osteoarthritis is inevitable and
cannot be avoided, even by this operation.33 This is
an expected consequence of the disease process
due to cartilage destruction. The main predictor of
healing or failure was the time elapsed between the
initial fracture and the treatment of the established
nonunion. A delay of 5 years or more would decrease
the success rate to only 62%.4
Complications of open repair include
hypertrophic scarring, avascular necrosis, carpal
instability, donor site pain, bone graft infection, screw
protrusion, and reflex sympathetic dystrophy.6 28 Due
to the cartilaginous surface and fracture healing
being an intraosseous process, open fixation of
a fractured scaphoid may further jeopardise the
blood supply of the scaphoid and drain away the
fracture haematoma.26 The radiocarpal ligaments
may also be damaged during open surgery.26 These
complications may be avoided using a minimally
invasive arthroscopic approach.6
Our centre’s practice
When a patient is admitted with the clinical suspicion of a scaphoid fracture (mechanism of
injury, tenderness at the anatomical snuffbox) but
no obvious fracture line seen on X-rays, we provide
the patient with a short-arm scaphoid cast and then
order a CT. The CT includes a coronal view to better
screen for any fracture. If no fracture is evident, a soft
splint is given for pain relief.
For an undisplaced non-comminuted waist
fracture, we offer conservative treatment with a
short-arm scaphoid cast for 6 weeks with interval
X-ray monitoring. Casting is kept for up to 4 to 8
weeks if the fracture line is still seen before surgery
is considered. We rarely perform surgery in this
patient group because we attained a 95% union rate
in compliant patients. In distal pole fractures, the
healing is generally quite good and we rarely resort
to excision.
We recommend surgery for all other types
of fracture alignment to prevent nonunion. We
generally use Herbert screws or cannulated screws,
subject to the surgeon’s preference. We almost
always use the percutaneous approach unless the
reduction cannot be obtained well. We use a volar
approach antegrade from the scaphoid tubercle
as we try to avoid the dorsal approach to prevent
cartilage injury. We, however, do use the dorsal
approach for proximal pole fractures. The success rate is about 95% for percutaneous fixations and 85
to 90% for open reductions.
For rehabilitation, we allow free mobilisation
on postoperative day 1 in waist fractures if the
fixation is rigid; we keep a slab for 2 weeks if the
fracture fixation is doubtful. We nevertheless allow
supervised gentle mobilisation for these fractures.
In proximal pole fractures, the purchase is usually
doubtful and we immobilise the wrist for 4 weeks.
In cases of nonunion, a plain MRI is helpful
for the assessment of avascular necrosis as these
conditions may require additional procedures such
as grafting. This condition is found in our hospital 12
to 18 times a year, and is always due to late referrals or
improper primary management including casting for
displaced waist fractures.
Conclusions
The current literature indicates no standard
treatment for scaphoid fractures. Different centres
have different approaches to the treatment of
scaphoid fractures and the evidence on which they
rely is controversial. However, the current trend is to
treat scaphoid fractures operatively, so as to limit the
number of days away from work and to allow patients
to regain function sooner.
References
1. Yin ZG, Zhang JB, Kan SL, Wang XG. Diagnosing suspected
scaphoid fractures: a systematic review and meta-analysis.
Clin Orthop Relat Res 2009;468:723-34. Crossref
2. Shenoy R, Pillai A, Hadidi M. Scaphoid fractures: variation in
radiographic views—a survey of current practice in the West
of Scotland region. Eur J Emerg Med 2007;14:2-5. Crossref
3. Adams JE, Steinmann SP. Acute scaphoid fractures. Orthop
Clin North Am 2007;38:229-35, vi. Crossref
4. Schuind F, Haentjens P, Van Innis F, Vander Maren C, Garcia-Elias M, Sennwald G. Prognostic factors in the treatment of
carpal scaphoid nonunions. J Hand Surg Am 1999;24:761-76. Crossref
5. Puopolo SM, Rettig ME. Management of acute scaphoid
fractures. Bull Hosp Jt Dis 2003;61:160-3.
6. Slade JF 3rd, Gillon T. Retrospective review of 234 scaphoid
fractures and nonunions treated with arthroscopy for union
and complications. Scand J Surg 2008;97:280-9.
7. Unay K, Gokcen B, Ozkan K, et al. Examination tests
predictive of bone injury in patients with clinically suspected
occult scaphoid fracture. Injury 2009;40:1265-8. Crossref
8. Brookes-Fazakerley SD, Kumar AJ, Oakley J. Survey of
the initial management and imaging protocols for occult
scaphoid fractures in UK hospitals. Skeletal Radiol
2009;38:1045-8. Crossref
9. Böhler L, Trojan E, Jahna H. The results of treatment of
734 fresh, simple fractures of the scaphoid. J Hand Surg Br
2003;28:319-31. Crossref
10. Cheung GC, Lever CJ, Morris AD. X-ray diagnosis of acute
scaphoid fractures. J Hand Surg Br 2006;31:104-9. Crossref
11. Ty JM, Lozano-Calderon S, Ring D. Computed tomography
for triage of suspected scaphoid fractures. Hand (N Y)
2008;3:155-8. Crossref
12. Ganapathi M, Savage R, Jones AR. MRI assessment of the
proximal pole of the scaphoid after internal fixation with a
titanium alloy Herbert screw. J Hand Surg Br 2001;26:326-9. Crossref
13. Wong TC, Yip TH, Wu WC. Carpal ligament injuries with
acute scaphoid fractures—a combined wrist injury. J Hand
Surg Br 2005;30:415-8. Crossref
14. Herbert TJ, Fisher WE. Management of the fractured scaphoid
using a new bone screw. J Bone Joint Surg Br 1984;66:114-23.
15. Geoghegan JM, Woodruff MJ, Bhatia R, et al. Undisplaced
scaphoid waist fractures: is 4 weeks’ immobilisation in a
below-elbow cast sufficient if a week 4 CT scan suggests
fracture union? J Hand Surg Eur Vol 2009;34:631-7. Crossref
16. Dias JJ, Wildin CJ, Bhowal B, Thompson JR. Should acute
scaphoid fractures be fixed? A randomized controlled trial. J
Bone Joint Surg Am 2005;87:2160-8. Crossref
17. Karantana A, Downs-Wheeler MJ, Webb K, Pearce CA,
Johnson A, Bannister GC. The effects of Scaphoid and
Colles casts on hand function. J Hand Surg Br 2006;31:436-8. Crossref
18. Hambidge JE, Desai VV, Schranz PJ, Compson JP, Davis TR, Barton NJ. Acute fractures of the scaphoid. Treatment by cast
immobilisation with the wrist in flexion or extension? J Bone
Joint Surg Br 1999;81:91-2. Crossref
19. Gelberman RH, Wolock BS, Siegel DB. Fractures and
non-unions of the carpal scaphoid. J Bone Joint Surg Am
1989;71:1560-5.
20. Kaneshiro SA, Failla JM, Tashman S. Scaphoid fracture
displacement with forearm rotation in a short-arm thumb
spica cast. J Hand Surg Am 1999;24:984-91. Crossref
21. Saedén B, Törnkvist H, Ponzer S, Höglund M. Fracture of the
carpal scaphoid. A prospective, randomised 12-year follow-up
comparing operative and conservative treatment. J Bone
Joint Surg Br 2001;83:230-4. Crossref
22. Moser VL, Krimmer H, Herbert TJ. Minimal invasive treatment
for scaphoid fractures using the cannulated herbert screw
system. Tech Hand Up Extrem Surg 2003;7:141-6. Crossref
23. Bhandari M, Hanson BP. Acute nondisplaced fractures of the
scaphoid. J Orthop Trauma 2004;18:253-5. Crossref
24. Dias JJ, Dhukaram V, Abhinav A, et al. Clinical and
radiological outcome of cast immobilisation versus surgical
treatment of acute scaphoid fractures at a mean follow-up of
93 months. J Bone Joint Surg Br 2008;90:899-905. Crossref
25. McQueen MM, Gelbke MK, Wakefield A, Will EM, Gaebler
C. Percutaneous screw fixation versus conservative treatment
for fractures of the waist of the scaphoid: a prospective
randomised study. J Bone Joint Surg Br 2008;90:66-71. Crossref
26. Yip HS, Wu WC, Chang RY, So TY. Percutaneous cannulated
screw fixation of acute scaphoid waist fracture. J Hand Surg
Br 2002;27:42-6. CrossRef
27. Shin AY, Hofmeister EP. Percutaneous fixation of stable scaphoid fractures. Tech Hand Up Extrem Surg 2004;8:87-94. Crossref
28. Naranje S, Kotwal PP, Shamshery P, Gupta V, Nag HL.
Percutaneous fixation of selected scaphoid fractures by
dorsal approach. Int Orthop 2010;34:997-1003. Crossref
29. Wu WC. Percutaneous cannulated screw fixation of acute
scaphoid fractures. Hand Surg 2002;7:271-8. Crossref
30. Polsky MB, Kozin SH, Porter ST, Thoder JJ. Scaphoid
fractures: dorsal versus volar approach. Orthopedics
2002;25:817-9.
31. Muller M, Germann G, Sauerbier M. Minimal invasive screw
fixation and early mobilization of acute scaphoid fractures
in the middle third: operative technique and early functional
outcome. Tech Hand Up Extrem Surg 2008;12:107-13. Crossref
32. Shih JT, Lee HM, Hou YT, Tan CM. Results of arthroscopic
reduction and percutaneous fixation for acute displaced
scaphoid fractures. Arthroscopy 2005;21:620-6. Crossref
33. Barton NJ. The late consequences of scaphoid fractures. J
Bone Joint Surg Br 2004;86:626-30. Crossref
34. Lindström G, Nyström A. Incidence of post-traumatic
arthrosis after primary healing of scaphoid fractures: a
clinical and radiological study. J Hand Surg Br 1990;15:11-3. Crossref
35. Amadio PC, Berquist TH, Smith DK, Ilstrup DM, Cooney
WP 3rd, Linscheid RL. Scaphoid malunion. J Hand Surg Am
1989;14:679-87. Crossref
36. Inoue G, Sakuma M. The natural history of scaphoid nonunion.
Radiographical and clinical analysis in 102 cases.
Arch Orthop Trauma Surg 1996;115:1-4. Crossref